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Although many psychoactive substances have been associated with violent behavior, only a subgroup manifests excessively aggressive behavior when sober or intoxicated. Theories to explain addictive behaviors in general may directly relate to the specific proclivity to exhibit excessive aggression in this subset of users. Certain personality traits and cognitive deficits coexist in individuals prone to both drug abuse and violence, suggesting a common origin. Because these excessive and compulsive behaviors have been linked with aberrations in the metabolism and activity of the neurotransmitters dopamine and serotonin, their origins may be partly genetic or biological. Alterations in neurotransmitter function influence activities within the brain's reward center to perturb nervous system arousal levels, thereby increasing stimulation-seeking behaviors. Manifestations of these neurobiological aberrations can be measured in physiological and biochemical processes that serve to mediate these behavioral and psychological outcomes.

For several decades, mainstream criminology has been dominated by sociological and political perspectives. Although findings from these fields must not be discarded or underplayed, considered alone, they do not offer a complete assessment of the contributions to criminal behavior. Data currently being generated from numerous behavioral sciences, such as behavioral genetics, physiological psychology, psychopharmacology, and endocrinology, indicate that biological factors play an equally significant role in the development of antisocial behavior and should be considered accordingly. Incorporation of the theoretical parameters and findings of these behavioral sciences into a criminological framework would yield valuable in formation regarding processes underlying antisocial behavior. Such a multidisciplinary approach is likely to enhance capabilities to predict, prevent, and manage antisocial behavior. Theoretical parameters, methodological issues, selected research findings, potential applications, and precautions are discussed.

AbstractDrawing on the social development model, the authors examine family risk and protective factors thought to influence problem behaviors among adolescents. They estimate the impact of family risk and protective factors on a variety of antisocial and health risk behaviors. Data are drawn from a sample of nearly 2,500 adolescents attending high‐risk schools in Trinidad and Tobago, a developing nation in the eastern Caribbean. The findings show that certain family risk factors play a more consistent role in shaping adolescent problem behaviors than others. In particular, adult history of antisocial behavior and parental attitudes favorable toward antisocial behavior and substance use emerge as the most robust risk factors. In accordance with previous research, family protective factors exerted only a minimal influence on behavioral outcomes. This finding confirms the need for additional theory and research on the protective factors that help reduce problem behavior among adolescents, particularly in developing nations.

The importance of basing public policy on sound scientific evidence is increasingly being recognized, yet many barriers continue to slow the translation of prevention research into legislative action. This work reports on the feasibility of a model for overcoming these barriers—known as the Research-to-Policy Collaboration (RPC). The RPC employs strategic legislative needs assessments and a rapid response Researcher Network to accelerate the translation of research findings into usable knowledge for policymakers. Evaluation findings revealed that this model can successfully mobilize prevention scientists, engage legislative offices, connect policymakers and experts in prevention, and elicit congressional requests for evidence on effective prevention strategies. On average, the RPC model costs $3,510 to implement per legislative office. The RPC can elicit requests for evidence at an average cost of $444 per request. The implications of this work, opportunities for optimizing project elements, and plans for future work are discussed. Ultimately, this project signals that the use of scientific knowledge of prevention in policymaking can be greatly augmented through strategic investment in translational efforts.

This paper reviews research suggesting that the prevention of intergenerational poverty will be enhanced if we add evidence-based family and school prevention programs to existing efforts to reduce poverty in order to address the adverse social environments that often accompany poverty. Government policies such as the Earned Income Tax Credit can reduce family poverty, but simply improving the economic stability of the family will not necessarily prevent the development of child and adolescent problems such as academic failure, antisocial behavior, drug abuse, and depression, all of which can undermine future economic wellbeing. We briefly review the evidence linking family poverty to adverse social environments, which can have deleterious effects on children's psychological, behavioral, neurological, and physical development. We then document the value of evidence-based family- and school-based prevention programs in effectively addressing these behavioral, emotional, cognitive, and neurophysiological factors that can put children at risk for continued poverty in adulthood. We also describe three family-based prevention programs that have been found to have a direct effect on families' future economic wellbeing. The evidence indicates that widely disseminating effective and efficient family- and school-based prevention programs can help to address both poverty itself and the effects of adverse social environments, making future poverty less likely. We conclude with specific recommendations for federal and state policymakers, researchers, and practitioners.

The prevalence of substance use behavior and the personal characteristics and social and cultural factors that influence substance use among children have been studied extensively. However, much less attention has been directed at the attitudes towards, and particularly the perceived risks of, using substances, and even less research has focused on Hispanic youth, whose rates of substance use have been increasing. Understanding what factors are associated with perceived risk is important because there is evidence that perceived risk is associated with subsequent substance use. The current study uses longitudinal data collected from 553 children in the Cicero Youth Development (CYD) Project to identify individual and environmental factors associated with the perceived risk of substance use. We pay particular attention to dimensions of nativity and acculturation in an effort to determine whether there are differences in risk perception between U.S.-born Hispanic children and their foreign-born counterparts. Results indicate that a number of factors are associated with perceived risk, including family support, parent-child communication, peer substance use, and child temperament. Additionally, U.S.-born Hispanics perceive substances as less risky than immigrants, and among immigrants, length of time in the U. S. is negatively associated with perceiving great risk in substance use. Implications for policy and prevention strategies are discussed.

Early adversity can alter development of neurocognition, including executive cognitive and emotional regulatory functions. This is the first study to explore differential relationships between personal (physical and emotional abuse and neglect, school and parental stressors) and community (neighborhood problems and witnessing neighborhood violence) stressors and neurocognition. Predominantly Latino children ( n = 553) aged 10 to 12 years completed tasks measuring intelligence, impulsivity, problem solving, cognitive flexibility, decision making, and emotion attributions. Adjusting for age and parent education, bivariate regression analyses found exposure to personal stressors to be associated with relative deficits in at least one neurocognitive function. Community stressors were related to relative deficits in emotion attributions and problem solving. In multivariate analyses, neglect was related to misattributions of emotion and IQ deficits, and physical abuse was related to problem solving. Community stressors were not correlated with neurocognition when viewed relative to personal stressors. Stressor types were differentially associated with performance on specific neurocognitive tasks.